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Search Results to Stavros Manolagas

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One or more keywords matched the following items that are connected to Manolagas, Stavros

Item TypeName
Academic Article Proteasomal degradation of Runx2 shortens parathyroid hormone-induced anti-apoptotic signaling in osteoblasts. A putative explanation for why intermittent administration is needed for bone anabolism.
Academic Article IL-6 is not required for parathyroid hormone stimulation of RANKL expression, osteoclast formation, and bone loss in mice.
Academic Article Connexin 43 is required for the anti-apoptotic effect of bisphosphonates on osteocytes and osteoblasts in vivo.
Academic Article Matrix-embedded cells control osteoclast formation.
Academic Article Estrogen receptor-a signaling in osteoblast progenitors stimulates cortical bone accrual.
Academic Article The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone.
Academic Article Osteocyte apoptosis is induced by weightlessness in mice and precedes osteoclast recruitment and bone loss.
Academic Article Divergent effects of selective peroxisome proliferator-activated receptor-gamma 2 ligands on adipocyte versus osteoblast differentiation.
Concept Bone Resorption
Academic Article Proposed standard nomenclature for new tumor necrosis factor family members involved in the regulation of bone resorption
Academic Article FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation.
Academic Article Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.
Academic Article PLEKHM1/DEF8/RAB7 complex regulates lysosome positioning and bone homeostasis.
Academic Article The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct.
Academic Article Deletion of FoxO1, 3, and 4 in Osteoblast Progenitors Attenuates the Loss of Cancellous Bone Mass in a Mouse Model of Type 1 Diabetes.
Academic Article Cxcl12 Deletion in Mesenchymal Cells Increases Bone Turnover and Attenuates the Loss of Cortical Bone Caused by Estrogen Deficiency in Mice.

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  • Bone Resorption